S2C). element and result in for CRC, may affect responsiveness to anti-VEGF. Chemically induced colitis (CIC) incis-Apc716/Smad4+/andcis-Apc716/Smad4+/KrasG12Dmice advertised development of colon tumors that were mainly resistant to anti-VEGF treatment. The myeloid growth element G-CSF was markedly improved in MOBK1B the serum after induction of colitis. Antibodies obstructing G-CSF, or its target Bv8/PROK2, suppressed tumor progression […]