This can be because of EPO-mediated expansion of RBCs thereby promoting pathogen replication whereas EPO could also induce anti-inflammatory effects thereby impairing the immune response towards plasmodia in the circulation

This can be because of EPO-mediated expansion of RBCs thereby promoting pathogen replication whereas EPO could also induce anti-inflammatory effects thereby impairing the immune response towards plasmodia in the circulation. with bacterias, linking HIF-1 expression to innate immune response [14] thus. Furthermore, pharmacological inhibition of PHDs, which initiates HIF-dependent gene appearance, effectively attenuates ischemia-reperfusion damage […]

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