These biochemical and functional studies support the idea that with protein folding/aggregation diseases, smaller subfibrillar particles may be much more pathological than larger amyloid fibrils or plaques [38]. commonalities between AD and CJD, and the newly emergent part of PrPCas a receptor for any oligomers. Keywords:Alzheimer’s disease, Amyloid, Prion, Receptor, Neurodegeneration == Intro == Worldwide, […]